Essence Equine

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Essence Equine Essence Equine was founded in February 2025 by Dr. Alyssa Keto out of love for equine veterinary medicine.
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She is passionate about providing services unique to horses in the field and brings years of experience in equine health, medicine, and lameness. Essence Equine, located out of Dover, NH, was founded in February 2025 by Dr. Alyssa Keto out of a deep-rooted love for equine veterinary medicine. Dr. Keto was raised on a small agricultural farm in rural Nebraska. She earned her Bachelor of Science in Agriculture with honors at Kansas State University in 2006. She then continued on to attend Kansas State University College of Veterinary Medicine, receiving her Doctorate of Veterinary Medicine in 2012. Dr. Keto completed a one-year internship at Myhre Equine Clinic and then continued on as an associate veterinarian there, focusing on ambulatory services. Dr. Keto is passionate about providing clients with the veterinary services unique to horses out in the field and brings with her years of experience in equine health, medicine, and lameness.

Wet, chilly autumn weather and thickening coats create ideal conditions for rain rot (dermatophilosis), one of the most ...
10/29/2026

Wet, chilly autumn weather and thickening coats create ideal conditions for rain rot (dermatophilosis), one of the most common skin conditions seen this time of year.

1. Cause: Dermatophilus congolensis, a gram-positive, non-acid-fast, facultatively anaerobic, branching filamentous actinomycete. Clinically normal, chronically infected horses are the main reservoir, and flare-ups follow wet weather. Moisture releases motile zoospores that are attracted to skin CO2 and invade compromised skin.

2. Pathogenesis: The infected epidermis hardens and lifts off as a crust, and wet crusts continue to release zoospores. Immunity after infection is not lasting, so it recurs, especially on farms with a history.

3. Signs: Raised crusty scabs with "paintbrush" tufts on the back, rump, neck, and face. Yellow-green pus is under larger crusts. Lower-leg lesions can occur at wet pasterns, especially on white legs, and rain rot is one cause of "scratches." Lesions can be painful, and some horses need sedation for crust removal.

4. Who's at risk: Young or immunosuppressed horses and those with concurrent illness or stress. Other risk factors are inadequate shelter, dense winter coats, and blankets that trap dampness.

5. Diagnosis: Rain rot is often recognized by its appearance. It is confirmed with cytology of the underside of a fresh crust, which shows "railroad tracks": 2-6 parallel rows of cocci under oil immersion. In atypical cases, biopsy shows branching hyphae divided in several planes along with coccoid cells and zoospores. Look-alikes, such as dermatophytosis (ringworm) and pemphigus foliaceus, need to be ruled out using culture or biopsy.

6. Treatment: The horse should be kept dry. Gently remove and dispose of crusts. Wash with an antimicrobial shampoo (2-4% chlorhexidine is the topical of choice), allow 10-15 minutes of contact time, rinse, and dry thoroughly. Many mild to moderate cases resolve with topicals and moisture control, while severe cases may need systemic antibiotics, such as trimethoprim-sulfamethoxazole.

7. Course: Once the skin stays dry, acute lesions often heal within about three weeks without scarring. Dry weather speeds healing.

8. Contagion and biosecurity: Direct contact, shared brushes, blankets, tack, and biting flies and ticks can spread it. Zoonotic infection is rare but is observed on the hands and arms if it occurs. Use separate tools, disinfect shared equipment, and wear gloves and wash with antimicrobial soap.

9. Prevention: Provide shelter, never blanket a wet horse, remove wet blankets, use breathable waterproof sheets, check under blankets, and control insects.

If you're a current client of Essence Equine and would like to discuss rain rot or other skin concerns for your horse, please reach out. If you would like to establish care for your horse(s) with us, please message or call us to schedule a new client appointment!

Autumn isn't a break from ticks here in New England. Adult blacklegged (deer) ticks (Ixodes scapularis) peak in October ...
10/27/2026

Autumn isn't a break from ticks here in New England. Adult blacklegged (deer) ticks (Ixodes scapularis) peak in October and November and can stay active on mild days well into the cold season.

1. Transmission – The blacklegged tick carries both Borrelia burgdorferi (Lyme disease) and Anaplasma phagocytophilum (anaplasmosis), and a single tick can carry both. Anaplasma can be transmitted in under 24 hours of attachment, while Lyme transmission risk climbs steeply after 48 hours. Daily checks and prompt removal matter for both.

2. Lyme disease – Many horses in New England test positive for exposure without ever showing signs, and a positive test alone does not confirm disease. The best-documented clinical forms are neuroborreliosis (infection of the nervous system), uveitis, and pseudolymphoma (skin nodules near the bite site). Shifting-leg lameness, stiffness, and cutaneous hyperesthesia (sensitivity to touch or grooming) are also reported, but are less well documented.

3. Anaplasmosis – This typically starts more acutely, with high fever, lethargy, a decreased appetite, distal limb and ventral edema, and reluctance to move. Some horses also show petechiae (small hemorrhages on the gums), icterus (jaundice), or ataxia. Bloodwork commonly shows mild anemia, leukopenia, and thrombocytopenia.

4. Testing – For Lyme disease, the Lyme Multiplex test measures antibodies to three outer surface proteins. OspA is associated with vaccination, though it can rise briefly early in infection. OspC appears about 3 weeks after infection and typically falls off within 4–5 months. OspF appears at 5–8 weeks and persists. For anaplasmosis, we can look for morulae (clusters of the organism) inside the neutrophils on a blood smear but always confirm with PCR. For either disease, results should be interpreted alongside clinical signs demonstrated by the horse.

5. Treatment – Both diseases are treated with a tetracycline-class antibiotic. Lyme disease is typically treated over a longer course (1+ month), while anaplasmosis is generally a two-week course, with patients responding quickly (often within 12–24 hours). Minocycline has substantially better oral bioavailability than doxycycline in horses, which is why it is the oral tetracycline of choice.

6. Daily tick checks – Ticks favor thin-skinned, protected areas: the mane, forelock, throatlatch, axillae (armpits), chest, ventral abdomen, groin, under the tail, and the heels. Run hands over these areas daily during grooming, especially after turnout near woods or in tall grass.

7. Removal – Grasp the tick close to the skin with fine-tipped tweezers and pull straight out with steady pressure. Avoid squeezing the body or applying substances to make it detach.

8. Prevention – Keep pasture edges and trails mowed, and limit brush along fence lines where ticks wait for a host. Repellents, such as permethrin-based products, are another option.

If you're a current client of Essence Equine and would like to discuss tick-borne disease testing or prevention for your horse, please reach out. If you would like to establish care for your horse(s) with us, please message or call us to schedule a new client appointment!

As horses shift from pasture to hay this fall, their water needs change. Grass is about 70–80% water, while hay is only ...
10/22/2026

As horses shift from pasture to hay this fall, their water needs change. Grass is about 70–80% water, while hay is only about 10–15%, so water intake has to make up the difference. Here's what we look at:

1. Daily water needs. A typical adult horse drinks about 5–10 gallons a day (about 1 gallon per 100 lb of body weight), and more with work, heat, or lactation. Horses drink roughly 1 gallon of water for every 3–4 pounds of hay (dry matter) they eat, so a hay-based diet drives water needs up. As pasture drops out of the diet, water intake from the bucket or trough needs to rise to keep up.

2. Why impaction happens. The large colon and cecum depend on adequate fluid to keep ingesta moving. When intake drops or the diet changes to dry forage, feed can dehydrate and pack, usually at the pelvic flexure of the large colon, where the colon narrows sharply and changes direction. This can cause a firm, dry impaction and a mild-to-moderate colic.

3. Risk factors. These include a sudden change from pasture to hay, poor dentition, reduced exercise, cooler weather that lowers drinking, water that is too cold or poorly accessible, and low-quality, coarse hay.

4. Signs to watch for. Decreased manure output, small, dry, or hard manure balls, reduced appetite, lethargy, mild abdominal discomfort, and looking at or nipping at the flank. Don't wait on a horse that stops passing manure.

5. Check hydration. Skin tent time, gum moisture, and capillary refill time can help you gauge it. Normally, capillary refill is under 2 seconds and a skin pinch on the neck or shoulder flattens within about 1–2 seconds. A slower skin tent or tacky gums suggests dehydration, though the skin tent is less reliable in older horses, whose skin loses elasticity. Gum color and refill time also help us assess how your horse is doing.

6. Monitor manure and gut sounds. Know what is normal for your horse's manure output and consistency, which makes changes easier to catch early. Most horses pass manure about 8–12 times a day, and gut sounds should be heard on both sides, upper and lower. Horses with a low manure count and reduced gut sounds warrant a call.

7. Support intake. Check water sources daily, keep buckets and troughs clean, and prevent freezing as the weather turns. Horses often prefer water in the 45–65°F range. Salt should always be available, and many horses need loose salt or a measured amount added to feed to drink well.

8. When to call us. Contact us right away if your horse is colicky, has stopped passing manure, is off feed, or seems depressed. Impactions can often be treated with fluids and supportive care when caught early, so a quick response matters.

If you're a current client of Essence Equine and would like to discuss hydration or colic prevention for your horse(s), please reach out. If you would like to establish care for your horse(s) with us, please message or call us to schedule a new client appointment!

As pastures wind down, hay becomes the foundation of your horse's diet. Not all hay is equal, and appearance alone won't...
10/20/2026

As pastures wind down, hay becomes the foundation of your horse's diet. Not all hay is equal, and appearance alone won't tell you what's in it. Here's what we look at when evaluating hay:

1. Maturity at cutting. Stage of maturity at harvest is the largest driver of nutritional value. As grass matures past the boot stage into heading and seed, lignin and fiber increase while protein and digestibility fall. Early-cut hay is higher in crude protein and digestible energy. Late-cut, stemmy hay is fine for easy keepers but may not meet the needs of hard keepers, seniors, or growing horses.

2. Forage analysis. Color and smell can't tell you nutrient content. A core sample from several bales of the same cutting, sent to a forage lab, will report:
- Crude protein (CP), often 8–14% in grass hays and higher in alfalfa.
- Acid detergent fiber (ADF), which reflects digestibility. Lower is more digestible.
- Neutral detergent fiber (NDF), which reflects how much a horse will voluntarily eat. Values above about 65% usually mean coarse, less palatable hay.
- Digestible energy (DE) and minerals, including the calcium-to-phosphorus ratio, ideally at least 1:1 to 2:1.
Most New England soils are low in selenium, so hay from the region is often deficient as well.

3. Sugar and starch (NSC). NSC is water-soluble carbohydrates (WSC) plus starch. For horses with equine metabolic syndrome (EMS), insulin dysregulation, PPID (Cushing's disease), or a history of laminitis, a common target is under 10–12% NSC on a dry matter basis. Soaking hay in cool water for 30–60 minutes and discarding the water can lower WSC, but how much varies widely by batch, and soaking also leaches minerals. Testing is the only way to know your starting point.

4. Mold and dust. Mold spores, including Aspergillus and thermophilic actinomycetes, along with fine dust, are major triggers of equine asthma (heaves) and may contribute to colic. Hay should smell sweet, with no dark, webbed, or powdery patches. Steaming reduces respirable particles and mold more effectively than soaking. Visibly moldy hay shouldn't be fed.

5. Foreign material and toxins. Check flakes for toxic weeds, sticks, wire, and animal remains. A carcass baled into hay, most often in round bales or haylage, can cause botulism. If you buy alfalfa shipped from outside the region, ask about blister beetles, whose toxin, cantharidin, can be fatal even in small amounts.

6. Moisture and storage. Hay baled above roughly 18–20% moisture is at risk of mold and heating. Monitor new hay for warmth over the first few weeks, and treat internal temperatures over 150°F as a fire risk. Store hay dry, off the ground on pallets, with good ventilation.

7. Transition slowly. A new batch of hay, like any change in forage, should be introduced over 7–10 days so the hindgut microbiome can adjust and colic risk stays low. Stocking enough from one consistent source means fewer abrupt changes.

If you're a current client of Essence Equine and would like to discuss your horse's hay or a forage analysis, please reach out. If you would like to establish care for your horse(s) with us, please message or call us to schedule a new client appointment!

Fall is a good time for a full wellness check, especially for senior horses. Small changes caught now are easier to mana...
10/15/2026

Fall is a good time for a full wellness check, especially for senior horses. Small changes caught now are easier to manage than problems found later in the season or during the winter. Here's what we focus on:

1. Body condition scoring (BCS). We score fat cover on a 1–9 scale over the ribs, loin, tailhead, withers, and behind the shoulder. Most horses do best at a 5–6/9. Seniors often lose topline and muscle before they lose fat, so we assess muscle separately from body condition.

2. Dentition. Sharp enamel points, hooks, wave mouth, loose or missing teeth, and diastemata (gaps where feed packs and causes periodontal disease) all affect how well a horse chews. Signs of a problem include quidding (dropping partially chewed feed), long fiber in the manure, slow eating, weight loss, and foul breath. Seniors may need to be supplemented with forage alternatives, such as soaked hay/alfalfa cubes or pellets, or a complete senior feed.

3. Musculoskeletal and hoof comfort. Arthritis and overall stiffness are common in older horses. We assess comfort, movement, and flexibility, and take a general look at the feet. If something concerns us, we'll recommend a closer exam or coordinate with your farrier. That helps us decide whether joint management, an anti-inflammatory plan, and/or an exercise adjustment might help keep them comfortable and moving.

4. PPID (Cushing's disease) screening. PPID is common in horses over 15, and early signs can be subtle. They include delayed or incomplete shedding (sometimes only small tufts of retained coat or long guard hairs), along with a long or curly/wavy coat, muscle loss over the topline, a pot belly, increased drinking and urination, recurrent infections, and laminitis. We screen with a resting (baseline) ACTH test. Fall is when ACTH naturally rises, so results are interpreted against seasonal reference ranges. For early or borderline cases, a TRH stimulation test is more sensitive. It measures ACTH before and after an injection of thyrotropin-releasing hormone (TRH), and it's best done outside the fall, since seasonal reference ranges aren't well established for it.

5. Bloodwork. A CBC (complete blood count) and serum chemistry panel give us a baseline for your horse's health. The CBC can show anemia, infection, or inflammation, while the serum chemistry panel covers kidney and liver values, protein levels, electrolytes, and muscle enzymes. Having that baseline matters more as horses age, or when they're on long-term medications such as NSAIDs (e.g., phenylbutazone or firocoxib) or pergolide, as it lets us catch changes early, often before any outward signs show up.

A fall exam is also a good time to review vaccines, parasite control, and your feeding plan as pasture turns over to hay.

If you're a current client of Essence Equine and would like to schedule a fall wellness or senior exam for your horse, please reach out. If you would like to establish care for your horse(s) with us, please message or call us to schedule a new client appointment!

Fall changes what's in your pasture, and that affects your horse's metabolism, gut, and weight. Here's what we look at a...
10/13/2026

Fall changes what's in your pasture, and that affects your horse's metabolism, gut, and weight. Here's what we look at as the season turns:

1. Cool nights and sunny days raise sugar. In cool weather, grasses keep making sugar during the day but use less of it at night to grow. As a result, nonstructural carbohydrate (NSC) levels, meaning sugars, starches, and fructans, can climb well above the 10–12% generally recommended for at-risk horses on cool, sunny days and after a frost. Fructans are the main concern in cool-season grasses, which is what most New England pastures are. They're stored mostly in the lower stem, or sheath, so short, closely grazed pasture often runs higher in sugar, not lower.

2. Grazing times matter. NSC is generally lowest in the early morning, before the sun has driven photosynthesis, and highest in the afternoon. After a frost, NSC can stay elevated until the grass thaws and the sun has been out for a few hours. Short, stressed, or overgrazed grass also runs higher in NSC.

3. Autumn flush. Fall rain after a dry summer can trigger a second burst of grass growth, and horses suddenly have lush, high-sugar grazing. Introduce it gradually, and limit turnout time for at-risk horses. Keep in mind that ponies and horses can eat about 1–1.5% of their body weight in pasture dry matter in just 3–4 hours of turnout, so limiting time alone doesn't fully control intake.

4. Who is most at risk? Horses and ponies with equine metabolic syndrome (EMS), insulin dysregulation, or PPID (Cushing's disease), plus any horse with a history of laminitis. Insulin-resistant horses can develop laminitis from hyperinsulinemia triggered by high-NSC grass. Early signs include a shortened stride, reluctance to turn, a "stuck" feeling on hard ground, increased digital pulses, and heat in the feet.

5. Use a grazing muzzle or drylot for at-risk horses. Muzzles can cut intake by roughly 30–80%, depending on fit and design, without eliminating turnout. A drylot with measured hay is the safest option for horses with a history of laminitis.

6. Pasture quality drops as the season goes on. After a killing frost, grass growth slows, protein and digestible energy fall, and fiber rises. Hard keepers and seniors may need extra calories before they start losing condition, and most horses will need hay in addition to grazing. Plan your transition to hay feeding early so you aren't scrambling when the pasture goes.

7. Make the change gradually. Any change in diet, including a move from pasture to hay, should take place over 7–10 days to let the gut microbiome adjust and reduce colic risk.

8. Know your horse's baseline. Monitor weight, body condition, and hoof health regularly so changes stand out early. If your horse has changed shape, gained fat in unusual places, or seems footsore, we can test for insulin dysregulation and PPID.

If you're a current client of Essence Equine and would like to discuss a pasture plan or testing for metabolic disease in your horses, please reach out. If you would like to establish care for your horse(s) with us, please message or call us to schedule a new client appointment!

Autumn is one of the most important times of the year for strategic parasite control. Here's how we approach it:1. F***l...
10/08/2026

Autumn is one of the most important times of the year for strategic parasite control. Here's how we approach it:

1. F***l egg count (FEC) first. An FEC is a f***l flotation that identifies and quantifies parasite eggs in manure. That includes strongyle-type eggs, ascarids (Parascaris spp., especially in young horses), and sometimes tapeworm eggs or other parasites. Strongyle eggs per gram (EPG) classify your horse as a low (under 200 EPG), moderate (200–500), or high (over 500) shedder. Roughly 20–30% of adult horses shed about 80% of the eggs in a herd, so blanket treatment of every horse isn't justified.

2. Anthelmintic resistance is widespread. Decades of interval deworming, where every horse was treated every eight (8) weeks regardless of need, have directly selected for resistant parasite populations. As a result, cyathostomins (small strongyles) now show broad resistance to fenbendazole and pyrantel, and resistance to the macrocyclic lactones (ivermectin and moxidectin) is emerging. Most healthy adults need only 1–2 targeted treatments a year based on shedding status.

3. Bots after a hard frost. A killing frost ends Gasterophilus fly activity, and a macrocyclic lactone (ivermectin or moxidectin) then clears the larvae from the stomach. Scrape any remaining bot eggs off the legs.

4. Tapeworms at least once a year. Anoplocephala perfoliata is associated with ileocecal intussusception, ileal impaction, and spasmodic colic, and it isn't reliably detected on a routine FEC. A fall praziquantel combination product covers it, and twice yearly (spring and fall) may be warranted on high-risk farms.

5. Encysted small strongyles. Cyathostomin larvae can stay encysted in the cecal and colonic mucosa through winter. Mass emergence (larval cyathostominosis) can cause acute diarrhea, hypoproteinemia, weight loss, and colic. Moxidectin (Quest, Quest Plus) is the primary product effective against encysted stages and is typically given in late fall or early winter. Ivermectin products, including ivermectin/praziquantel combinations like Equimax and Zimecterin Gold, are effective against adult small strongyles but not the encysted larvae. Moxidectin isn't recommended for foals under six (6) months or for thin, debilitated horses without veterinary guidance.

6. Confirm efficacy. A f***l egg count reduction test (FECRT), which is a repeat FEC 10–14 days after treatment, tells us whether the product is working on your farm. A reduction below 90–95% suggests resistance.

For an accurate sample, collect 2–3 fresh f***l balls (less than 12 hours old), seal them in a labeled bag, refrigerate them (don't freeze), and get them to us (or your veterinarian) within 24–48 hours.

Pasture and turnout management is where most parasite control fails, and no dewormer can make up for it. Strongyle larvae develop in the environment and can survive for months, so that's where most of the parasite population actually lives. Pick up manure in paddocks and small turnouts at least twice weekly. That removes eggs before larvae can develop and migrate onto grass. Avoid overstocking, because crowded horses graze closer to manure piles. Where possible, rest or rotate pastures, and mow or harrow them only during hot, dry weather. Horses on drylots aren't exempt. Larvae survive less well on bare dirt, but manure builds up fast in a small space and horses pick up larvae eating hay off the ground. Pick up drylot manure daily if you can, and feed hay in a feeder, slow feeder, or on a mat rather than directly on the dirt.

If you'd like to set up f***l egg counts or build a deworming plan for your horses, message or call us!

Going into a New England winter, the real question isn't "blanket or no blanket." It's whether your horse has the body c...
10/06/2026

Going into a New England winter, the real question isn't "blanket or no blanket." It's whether your horse has the body condition and forage intake to thermoregulate, and whether you can still assess that condition under a blanket. Here's what we look at every fall:

1. Body condition score (BCS). We use the Henneke scale, which runs from 1 (emaciated) to 9 (extremely obese). Most horses should go into winter at a 5, and hard keepers and seniors at a 5–6, so they have some fat reserve to draw on. Score with your hands, not your eyes, because a winter coat can hide a full point or more. Palpate fat cover over the ribs, withers, loin, tailhead, neck crest, and behind the shoulder. At a 5, the ribs are easily felt but not seen and the back is level.

2. Blankets mask weight loss. Take the blanket all the way off at least once a week and palpate. We routinely see horses lose 1–2 condition scores over the winter without the owner noticing until spring. Body fat is insulation and an energy reserve, so a thin horse has less of both right when it needs them most.

3. Lower critical temperature (LCT). LCT is the temperature below which a horse has to spend extra energy to maintain core body temperature. An adult horse that is dry and has a full winter coat has an LCT of roughly 18°F, and closer to 40–50°F if clipped or short-coated. For every degree below LCT, energy requirements rise by about 1%. A 0°F night can increase a horse's maintenance energy needs by around 20%.

4. Forage is the heater. Microbial fermentation of fiber in the hindgut produces far more metabolic heat than digestion of grain. Feed at least 1.5–2% of body weight in forage per day, which is 15–20 lbs of hay for a 1,000 lb horse, and increase it as temperatures drop. Free-choice hay on cold nights is ideal. Water intake matters just as much: horses drink less in cold weather, which raises the risk of impaction colic. Keep water ice-free and ideally 45–65°F.

5. Wet plus wind is the real danger. A winter coat insulates by trapping air through piloerection, meaning the hair stands up. A soaked or flattened coat loses most of that insulating value, and wind strips heat away much faster. Shelter from wind and precipitation often matters more than a blanket.

6. Horses that usually do need a blanket: clipped horses, seniors with reduced body reserves, horses at a BCS of 4 or below, horses with PPID (Cushing's) or an abnormal coat, horses without adequate shelter, and horses not yet acclimated after coming from a warmer climate.

7. If you blanket, do it right. Check fit at the withers, shoulders, and points of hip, and look for rubs and pressure sores. Check under the blanket for dermatophilosis (rain rot), and pull a soaked or torn blanket right away. Don't overblanket. A horse that sweats under a heavy blanket ends up with a wet coat, which leaves it colder than one that was never blanketed.

8. Rule out underlying causes of weight loss. If a horse can't hold condition on the same forage, look at dentition (sharp enamel points, malocclusions, loose or missing teeth), parasite burden, PPID, and gastrointestinal disease. Many of these can be picked up on a fall exam and bloodwork.

If you'd like help condition scoring your horse or working out a winter feeding and blanketing plan, message or call us. We're happy to help!

Fall is one of the highest-risk times of the year for plant toxicity in horses. Pastures thin out, storms bring down bra...
10/01/2026

Fall is one of the highest-risk times of the year for plant toxicity in horses. Pastures thin out, storms bring down branches and leaves, and horses that would normally ignore a plant start browsing whatever they can reach. These are the plants we worry about most in New England this time of year:

1. Red maple (Acer rubrum). Wilted or dried leaves are the problem, not fresh ones, so fallen leaves and storm-downed branches are the main risk. The leaves stay toxic for about 30 days. Gallic acid and its metabolites cause oxidative damage to red blood cells, which leads to methemoglobinemia, Heinz body formation, and severe hemolytic anemia. As little as 1.5 lbs of dried leaves can be toxic to a 1,000 lb horse, and roughly double that can be fatal. Signs usually appear within 1–5 days: lethargy, anorexia, brown or muddy mucous membranes, jaundice, dark red-brown urine, and elevated heart and respiratory rates. Secondary kidney injury is common. There's no antidote, so treatment is aggressive supportive care, IV fluids, and sometimes blood transfusion.

2. Wild cherry (black cherry and chokecherry, Prunus spp.). The leaves contain cyanogenic glycosides. When leaves wilt, whether from frost, drought, or broken branches, those compounds break down into hydrogen cyanide. Cyanide blocks the cells' ability to use oxygen, so blood stays bright cherry-red even as tissues starve. Signs can progress within minutes to hours: bright red mucous membranes, labored breathing, anxiety, trembling, collapse, and sudden death.

3. Oak and acorns (Quercus spp.). Hydrolyzable tannins (gallotannins) cause GI ulceration and renal tubular damage. Signs include colic, constipation followed by diarrhea (sometimes bloody), edema, and elevated kidney values on bloodwork. Heavy acorn drops after storms or wind are the biggest risk, and some horses actively seek them out.

4. Yew (Taxus spp.). It's common in landscaping and one of the most toxic plants a horse can eat. Taxine alkaloids block sodium and calcium channels in the heart, causing bradycardia, arrhythmias, and cardiac arrest. Roughly a pound of clippings can kill a 1,000 lb horse, and often much less. Many horses are simply found dead, sometimes within hours of eating it. Never toss hedge clippings into a paddock, and ask neighbors not to either.

5. Black walnut (Juglans nigra). Bedding with as little as 20% black walnut shavings can cause signs, usually within 8–24 hours: distal limb edema, heat in the feet, increased digital pulses, and laminitis. Remove the bedding immediately and hose the legs. Know where your shavings come from.

6. White snakeroot (Ageratina altissima). It grows in shaded, wooded edges and blooms late summer into fall. Its toxin, tremetol, is cumulative, so repeated small exposures add up. It damages heart and skeletal muscle, causing trembling, weakness, reluctance to move, and signs of heart failure.

Prevention is easy. Walk your fence lines after every storm, remove downed branches and leaf piles, identify the trees along and over your pastures, and keep plenty of hay out so horses aren't forced to browse.

If you think your horse may have eaten any of these, call us right away. Early treatment makes a significant difference!

Equine emergencies don't always present dramatically. A horse that's down, thrashing, or actively hemorrhaging is obviou...
09/29/2026

Equine emergencies don't always present dramatically. A horse that's down, thrashing, or actively hemorrhaging is obvious. Early colic, choke, synovial wounds, or systemic illness can start subtly, with partial inappetence, reduced manure output, a change in stance, or a mild lameness that progresses over hours.

A few things that matter clinically:

1. Know normal vitals. For an adult horse at rest, heart rate is roughly 28-44 beats per minute, respiratory rate is 10-24 breaths per minute, and re**al temperature is about 99.0-101.0°F. Mucous membranes should be moist and pink, with a capillary refill time < 2 seconds. A persistently elevated heart rate, tacky or discolored gums, or prolonged capillary refill can point to pain, dehydration, or poor perfusion, and those findings help us to triage over the phone.

2. Call early for these: colic signs (flank watching, pawing, rolling, reduced gut sounds and/or manure); choke (feed material or saliva at the nostrils or from the mouth, repeated stretching of the neck); any wound near a joint, tendon sheath, or eye; non-weight-bearing lameness; respiratory distress; and fever with depression. Synovial involvement and strangulating lesions are time-sensitive, and outcomes depend heavily on how quickly they're recognized and treated.

3. Stabilize without making it worse. Pull feed and water if colic or choke is suspected, unless you’re directed otherwise. Don't give medications before you’ve discussed it with us, as an anti-inflammatory can sometimes mask pain and change how exam findings are interpreted. Apply steady and direct pressure to bleeding with a clean bandage material, and don't probe or flush a wound near a joint. Don't force a horse that can't bear weight to walk or load.

4. Have a plan before you need it. Know how to reach us after hours, have a working trailer and a driver lined up, and think through where your horse would go if surgical referral is needed. Those decisions are much harder to make at midnight.

When your horse needs us, we come to you. If you're looking at something and aren't sure whether or not it's an emergency, message or call us. An early call is always better than a late one!

Address

191 Emerald Lane
Dover, NH
03820

Opening Hours

Monday 9am - 5pm
Tuesday 9am - 5pm
Wednesday 9am - 5pm
Thursday 9am - 5pm
Friday 9am - 5pm

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