MaQzee Tech

24/11/2025

Hello!
dear family and friends,
good to see you again....
@

30/08/2025

Another update is about to launch on the 1st of September, so you all should be ready... I'm not forcing anyone to do it but if you are interested, just wait till that time.
withdrawal are combined.
Good luck.

29/08/2025

New Free USDT site Like Labubu

Payment confirmed for free so rush am now
Registration Link

https://dogs-1.online/register?code=82AE4DFO

Start Quantification or Click to Claim daily earnings
Commission available
Minimum Withdrawal $2

Rush it now

28/05/2023

Conservation
We care!.....

01/06/2022

27/05/2022

Animals can also be part of it.
They are essential too.
Treat them with respect......
....

01/12/2021

Cont.
CANINE PARVOVIRUS ETIOLOGY AND PATHOPHYSIOLOGY
Virus is shed in the f***s of infected dogs within 4–5 days of exposure (often before clinical signs develop), throughout the period of illness, and for ~10 days after clinical recovery. Infection is acquired through direct oral or nasal contact with virus-containing f***s or indirectly through contact with virus-contaminated fomites (eg, environment, personnel, equipment). Viral replication occurs initially in the lymphoid tissue of the oropharynx, with systemic illness resulting for subsequent hematogenous dissemination. CPV preferentially infects and destroys rapidly dividing cells of the small-intestinal crypt epithelium, lymphopoietic tissue, and bone marrow. Destruction of the intestinal crypt epithelium results in epithelial necrosis, villous atrophy, impaired absorptive capacity, and disrupted gut barrier function, with the potential for bacterial translocation and bacteremia.
Lymphopenia and neutropenia develop secondary to destruction of hematopoietic progenitor cells in the bone marrow and lymphopoietic tissues (eg, thymus, lymph nodes, etc) and are further exacerbated by an increased systemic demand for leukocytes. Infection in utero or in pups

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01/12/2021

CANINE PARVOVIRUS
Etiology and Pathophysiology.
Canine parvovirus (CPV) is a highly contagious and relatively common cause of acute, infectious GI illness in young and/or unvaccinated dogs. Although its exact origin is unknown, it is believed to have arisen from feline panleukopenia virus. It is a nonenveloped, single-stranded DNA virus, resistant to many common detergents and disinfectants, as well as to changes in temperature and pH. Infectious CPV can persist indoors at room temperature for at least 2 months; outdoors, if protected from sunlight and desiccation, it can persist for many months and possibly years. In North America, clinical disease is largely attributed to CPV-2b; however, infection with a newer and equally virulent strain, CPV-2c, is increasingly common, having been identified in at least 15 states. To date, no association has been identified between CPV strain and severity of clinical disease.
Young (6 week to 6 month old), unvaccinated or incompletely vaccinated dogs are most susceptible. Breeds described as at increased risk include:
1. Rottweilers
2. Doberman Pinschers
3. American Pit Bull Terriers
4. English Springer Spaniels
5. German Shepherds
Assuming sufficient colostrum ingestion, puppies born to a dam with CPV antibodies are protected from infection for the first few weeks of life; however, susceptibility to infection increases as maternally acquired antibody wanes. Stress (eg, from weaning, overcrowding, malnutrition, etc), concurrent intestinal parasitism, or enteric pathogen infection (eg, Clostridium spp, Campylobacter spp, Salmonella spp, Giardia spp, coronavirus) have been associated with more severe clinical illness. Among dogs >6 months old, intact male dogs are more likely than intact female dogs to develop CPV enteritis.

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